By Laura Nieves (auth.), George Y. Wu, Nathan Selsky, Jane M. Grant-Kels (eds.)
Atlas of Dermatological Manifestations of Gastrointestinal Disease offers a finished compendium of digestive tract ailments with dermatological manifestations. The paintings is prepared through digestive signs, with sections according to the categorical digestive ailment etiology. every one part comprises paired chapters of textual content, one on gastrointestinal manifestations and the opposite on dermatological manifestations. The atlas comprises scientific displays, pathophysiology, differential prognosis, diagnostic tests/procedures, pathology, and references provided with massive numbers of pictures.
Concise and straightforward to exploit, Atlas of Dermatological Manifestations of Gastrointestinal illness is a big source for gastroenterologists, dermatologists, and internists alike within the remedy of digestive tract diseases.
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Extra info for Atlas of Dermatological Manifestations of Gastrointestinal Disease
1)  • Generalized: acrofacial, vulgaris, mixed  • Universal: >80% of the body  • Ponctué: rare, discrete, confetti macules  • Trichrome: tan zone of varying width between normal and depigmented skin  • Quadrichrome: additional marginal or perifollicular hyperpigmentation  • Blue vitiligo: blue-gray hue  • Inflammatory: erythema at border of depigmented macules  L. edu J. J. M. 9% of persons with vitiligo (13fold increase over the general population)  Vitiligo probably represents a heterogeneous group of disorders with multifactorial pathogenesis, including: • Loss of functioning epidermal and sometimes hair follicle melanocytes  • Current hypothesis is that vitiligo is a group of heterogeneous pathophysiologic disorders with a similar phenotype; although systemic autoimmune processes may underlie generalized vitiligo, more localized processes (dysregulation of the nerve system or cytotoxic stress) may underlie focal segmental or focal vitiligo [ 1 ] Histopathological features include: • Few or no melanocytes within the lesion; melanocytes on the edge of the lesion are large, with long dendritic processes filled with melanin (see Fig.
3. Merger BS, Merger D. Carcinoma of the esophagus and tylosis: a lethal genetic combination. Cancer. 1993;72:17–9. 4. Howel-Evans W, McConnell RB, Clarke CA, Sheppard PM. Carcinoma of the oesophagus with keratosis palmaris et plantaris (tylosis). Q J Med. 1958;27:413–9. L. edu J. J. M. Y. Wu et al. 1007/978-1-4614-6191-3_6, © Springer Science+Business Media New York 2013 13 14 Fig. 1 Leukoplakia. Shaggy white plaque on the lateral tongue Fig. 2 Focal keratoderma of the plantar foot L. Zakko et al.
9 Epidermolysis Bullosa Acquisita: Gastrointestinal Features Liam Zakko Epidermolysis bullosa acquisita is an inherited connective tissue disease that causes blistering of the skin and mucous membranes. The incidence is estimated at 1/50,000, with severity ranging from mild to debilitating. 5/million persons/year, with increased incidence in persons of sub-Saharan African descent. Mean age of onset is 50. The gastrointestinal (GI) symptoms associated with this disease include [1, 2]: • Dysphagia, chronic constipation, fecal impaction Gastrointestinal clinical signs and findings include [1–4]: • Mucosal involvement in 20% of cases: mostly oral but also laryngeal and conjunctival • Esophagus can have blisters, erosions, scars, webs, ulcers, and strictures (see Fig.